czym rozni si ew skutecznosci stary GTF od nowego chelatu chromu?

czy nowy jest duzo skuteczniejszy od stArgo chromu GTF?
jakie inne suplr polecacie przy diecie wysokoweglowej?

jak to w przyrodzie nowe /lepsze zastepuje stare

a czym sie rozni niech odpowie producent

Chela-Chrom™ to suplement diety zawierający najlepiej przyswajalną i najbardziej bezpieczną formę chromu organicznego pod postacią chelatu aminokwasowego ALBIONź

dlaczego preferujesz diete wysokoweglowodanowa ?

osobiscie lubie diete ODPOWIEDNIOweglowodanowa :D

taki off top - ktory enzym albo cokolwiek w organizmie porzebuje chromu do swojej aktywnosci?

ot taki ciekawy przypadki

Chromium deficiency after long-term total parenteral nutrition.
Brown RO, Forloines-Lynn S, Cross RE, Heizer WD.

A 63-year-old female developed unexplained hyperglycemia and glycosuria during administration of a total parenteral nutrition regimen on which she had been stable for several months. Because the patient had no history of diabetes or evidence of an infection, chromium deficiency was considered. Plasma chromium level was 0.1 microgram/dl (laboratory reference interval: 1.8-3.8 micrograms/dl). Fourteen days of supplemental intravenous chromium chloride (200 micrograms/day) allowed complete withdrawal of exogenous insulin with no further hyperglycemia or glycosuria. Correction of unexplained glucose intolerance following vigorous chromium supplementation indicates that the patient had chromium deficiency. Subsequent plasma chromium levels remained unchanged, possibly reflecting the sensitivity limits of the assay that was used, the uncertainty that exists regarding appropriate reference intervals for this element, and the fact that plasma levels do not always correlate with total body stores. The patient did not manifest peripheral neuropathy, which was present in one of the two previously reported cases, nor encephalopathy, which was reported in the other. We conclude that this patient developed chromium deficiency as a result of inadequate administration of chromium in the parenteral formula (6 micrograms/day) plus excessive enteric losses, and she presented with glucose intolerance as the only clinical manifestation of the deficiency. Caution should be exercised when interpreting plasma chromium in patients with suspected deficiency.



Chromium deficiency, glucose intolerance, and neuropathy reversed by chromium supplementation, in a patient receiving long-term total parenteral nutrition.
Jeejeebhoy KN, Chu RC, Marliss EB, Greenberg GR, Bruce-Robertson A.

A white female, now age 40 and receiving total parenteral nutrition for more than 5 years, developed unexpected 15% weight loss after 3 1/2 years of regimen, together with peripheral neuropathy confirmed by nerve conduction measurements. An intravenous glucose tolerance test showed that the fractional rate (K) had decreased to 0.89%/min (normal greater than 1.2). There was observed during this glucose infusion a borderline normal insulin response with a fall in plasma free fatty acids and in plasma leucine. During daily infusion of well over 400 g of glucose, the respiratory quotient was 0.66. Chromium balance was negative. Chromium levels were, in blood 0.55 ng/ml (normal 4.9 to 9.5) and in hair 154 to 175 ng/g (normal greater than 500). Regular insulin daily (45 micron) in the infusate nearly maintained euglycemia but despite this, and even with further glucose intake to restore weight loss, intravenous glucose tolerance test (K) and respiratory quotient were unchanged. Administration of insulin was then stopped and 250 microng of Cr added to the daily total parenteral nutrition infusate for 2 weeks. After this the intravenous glucose tolerance test (K) and respiratory quotient became normal (1.35 and 0.78, respectively). Over the next 5 months insulin was not needed and glucose intake had to be reduced substantially to avoid overweight. In this period nerve conduction and well-being returned to normal. With a maintenance addition of chromium to the total parenteral nutrition infusate (tentatively this addition is 20 microng/day) the patient has remained well for 18 months (to July 1976). These results suggest that relatively isolated chromium deficiency in man, hitherto poorly documented, causes 1) glucose intolerance, 2) inability to utilize glucose for energy, 3) neuropathy with normal insulin levels, 4) high free fatty acid levels and low respiratory quotient and, 5) abnormalities of nitrogen metabolism.



i cos wiecej


Clinical and biochemical aspects of chromium deficiency.
Wallach S.

The essentiality of chromium (Cr) in animal and human nutrition is now well accepted. In animals, Cr deficiency can cause a diabetic-like state, impaired growth, elevated blood lipids, increased aortic plaque formation, and decreased fertility and longevity. The ability of Cr to potentiate insulin sensitivity has considerable experimental support. In the human, Cr deficiency has been demonstrated unequivocally in only one clinical situation, patients on total parenteral nutrition without added Cr. In such patients, impaired glucose tolerance, hyperglycemia, relative insulin resistance, peripheral neuropathy, and a metabolic encephalopathy have been noted with reversal of the clinical phenomena by Cr repletion. Many studies have been performed to determine whether Cr deficiency may be important in other clinical conditions, namely, diabetes mellitus, pregnant and parous women, and the aged population. Available data indicate that Cr supplementation can improve glucose metabolism in glucose intolerant individuals and decrease the total/HDL cholesterol ratio regardless of the status of glucose tolerance. However, whether Cr supplementation has long-term health benefits is unknown. Further, despite many tantalizing observations, it is still unclear whether Cr deficiency, latent or overt, is common in any human situation other than generalized malnutrition and total parenteral nutrition without added Cr. Technical uncertainties in the analysis of Cr, Cr contamination of food by the use of stainless steel processing equipment and eating utensils, and the lack of a clinically feasible test for Cr deficiency continue to impede progress in Cr research. Nevertheless, there is considerably more clarity as to plasma and urine Cr levels, food and tissue Cr content, and metabolic pathways of Cr metabolism than existed a decade ago. It is expected that progress will accelerate, since critical questions can now be addressed regarding the role of Cr in human nutrition.

yyy a mozesz przetmulaczyc w skrocie?

kurcze mam obled

ale na szybko

niedobor moze prowadzic do:
1) nietolernacja glukozy ,
2) niemoznosc wykorzystania glukozy jako paliwa
3) choroby ukladu nerwowego przy normalnym poziomie insuliny (tego nie kumam ale jestem po 14 godzinach roboty :D)
4) wysoki poziom wolnych kwasow tluszczowych
5) nieprawidlowosci w metabolizmie azotu


sorry jesli cos pokrecilem
alem lekko zacmiony :D

Zmieniony przez - Biniu w dniu 2008-11-27 20:56:50

dzieki, ciekawe to
ale ciekawy jestem co chrom takiego robi? z jakim bialkiem oddzialywuje w metabolizmie ze ma az taki na organizm wplyw?

a to juz nie moja dzialka - Slawek Ambroziak sledzi szlaki - ja jedynie czasem rzucam okiem jesli cos mi nie chce sie spasowac z reszta wiedzy :D

chrom w obrazie makro wychodzi mi ze dziala wiec nie zaglebialem sie dalej